Unidad de Neurobiología
CTS562
Harvard University
Cambridge, Estados UnidosPublications en collaboration avec des chercheurs de Harvard University (7)
2020
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Increased mitochondrial calcium levels associated with neuronal death in a mouse model of Alzheimer’s disease
Nature Communications, Vol. 11, Núm. 1
2012
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Reducing available soluble β-amyloid prevents progression of cerebral amyloid angiopathy in transgenic mice
Journal of Neuropathology and Experimental Neurology, Vol. 71, Núm. 11, pp. 1009-1017
2010
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Triflusal reduces dense-core plaque load, associated axonal alterations and inflammatory changes, and rescues cognition in a transgenic mouse model of Alzheimer's disease
Neurobiology of Disease, Vol. 38, Núm. 3, pp. 482-491
2009
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Matrix metalloproteinase inhibition reduces oxidative stress associated with cerebral amyloid angiopathy in vivo in transgenic mice
Journal of Neurochemistry, Vol. 109, Núm. 6, pp. 1636-1647
2007
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Antibody-mediated clearance of amyloid-β peptide from cerebral amyloid angiopathy revealed by quantitative in vivo imaging
Journal of Neuroscience, Vol. 27, Núm. 8, pp. 1973-1980
2006
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Characterization of amyloid deposition in the APPswe/PS1dE9 mouse model of Alzheimer disease
Neurobiology of Disease, Vol. 24, Núm. 3, pp. 516-524
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Kinetics of cerebral amyloid angiopathy progression in a transgenic mouse model of Alzheimer disease
Journal of Neuroscience, Vol. 26, Núm. 2, pp. 365-371