Publicacións (23) Publicacións de ANTONIO ROSAL VELA

2023

  1. Endothelial progenitor cells as biomarkers of diabetes-related cardiovascular complications

    Stem Cell Research and Therapy, Vol. 14, Núm. 1

  2. PCR-Based Strategy for Introducing CRISPR/Cas9 Machinery into Hematopoietic Cell Lines

    Cancers, Vol. 15, Núm. 17

2022

  1. Assessment of endothelial colony forming cells delivery routes in a murine model of critical limb threatening ischemia using an optimized cell tracking approach

    Stem Cell Research and Therapy, Vol. 13, Núm. 1

2021

  1. Long term response to circulating angiogenic cells, unstimulated or atherosclerotic pre-conditioned, in critical limb ischemic mice

    Biomedicines, Vol. 9, Núm. 9

2020

  1. Atherosclerotic pre-conditioning affects the paracrine role of circulating angiogenic cells ex-vivo

    International Journal of Molecular Sciences, Vol. 21, Núm. 15, pp. 1-21

  2. Identification of the initial molecular changes in response to circulating angiogenic cells-mediated therapy in critical limb ischemia

    Stem Cell Research and Therapy, Vol. 11, Núm. 1

  3. Molecular signatures of atherosclerotic plaques: An up-dated panel of protein related markers

    Journal of Proteomics, Vol. 221

  4. REX-001, a BM-MNC Enriched Solution, Induces Revascularization of Ischemic Tissues in a Murine Model of Chronic Limb-Threatening Ischemia

    Frontiers in Cell and Developmental Biology, Vol. 8

2019

  1. Ifnγ induces epigenetic programming of human t-bethi b cells and promotes tlr7/8 and il-21 induced differentiation

    eLife, Vol. 8

2018

  1. CD38 promotes pristane-induced chronic inflammation and increases susceptibility to experimental lupus by an apoptosis-driven and TRPM2-dependent mechanism

    Scientific Reports, Vol. 8, Núm. 1

2017

  1. Human canonical CD157/Bst1 is an alternatively spliced isoform masking a previously unidentified primate-specific exon included in a novel transcript

    Scientific Reports, Vol. 7, Núm. 1

  2. Increased expression of microRNA-155 in peripheral blood mononuclear cells from psoriasis patients is related to disease activity

    Journal of the European Academy of Dermatology and Venereology, Vol. 31, Núm. 2, pp. 312-322

2016

  1. Identification of multiple transferrin species in the spleen and serum from mice with collagen-induced arthritis which may reflect changes in transferrin glycosylation associated with disease activity: The role of CD38

    Journal of Proteomics, Vol. 134, pp. 127-137

  2. Supporting data for the MS identification of distinct transferrin glycopeptide glycoforms and citrullinated peptides associated with inflammation or autoimmunity

    Data in Brief

2015

  1. Distinct serum proteome profiles associated with collagen-induced arthritis and complete Freund's adjuvant-induced inflammation in CD38-/- mice: The discriminative power of protein species or proteoforms

    Proteomics, Vol. 15, Núm. 19, pp. 3382-3393

2013

  1. Increased CD38 expression in T cells and circulating anti-CD38 IgG autoantibodies differentially correlate with distinct cytokine profiles and disease activity in systemic lupus erythematosus patients

    Cytokine, Vol. 62, Núm. 2, pp. 232-243

  2. Proteomic analysis (2d-dige and ms/ms) of sera and lymphoyd organs from collagen-induced arthritic mice

    Proteómica: revista de la Sociedad Española de Proteómica, Núm. 9, pp. 109-109

2012

  1. El análisis proteómico mediante 2D-DIGE y MS/MS permite discriminar en un mismo experimento entre ratones silvestres (WT) o deficientes CD38 (CD38ko) y entre ratones afectados o no por la artritis inducida por colágeno

    Proteómica: revista de la Sociedad Española de Proteómica, Núm. 8, pp. 117-117

  2. Increased expression and phosphorylation of the two S100A9 isoforms in mononuclear cells from patients with systemic lupus erythematosus: A proteomic signature for circulating low-density granulocytes

    Journal of Proteomics, Vol. 75, Núm. 6, pp. 1778-1791

  3. Mice deficient in CD38 develop an attenuated form of collagen type II-induced arthritis

    PLoS ONE, Vol. 7, Núm. 3